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			== Semi-protected edit request on 10 May 2022 - Genetics of Pneumonia ==
	== Edit Request - Cost ==
			{{edit semi-protected\|Pneumonia\|answered=yes}}

			It has been found that vulnerability to Pneumonia and its severity may be linked to underlying genetic mechanisms. These mechanisms, such as the ] (CYP1A1) regulating ] and ], <ref>Guin, Debleena, et al. “Human Genetic Factors Associated with Pneumonia Susceptibility, a Cue for Covid-19 Mortality.” MedRxiv, Cold Spring Harbor Laboratory Press, 1 Jan. 2021, https://www.medrxiv.org/content/10.1101/2021.06.03.21258106v1.full. </ref> can influence the efficacy of the immune system which can either be detrimental or beneficial in combating Pneumonia.
	Mohamad Christi created a cost-effective invention to help children with pneumonia in low-income countries. It works similarly to a bubble-cPAP, however, it is made out of a simple shampoo bottle filled with water and some tubing attached. This simple invention cut the price of treatment by approximately 90% which helped parents in low-income countries who could not afford bubble-cPAP treatment for their children with pneumonia. <ref>{{cite web \|title=Health Care \|url=https://www.economist.com/science-and-technology/2018/09/08/how-a-shampoo-bottle-is-saving-young-lives \|website=The Economist \|accessdate=11 September 2018}}</ref>

			Individuals with certain genetic variants can be at risk for a higher susceptibility to Pneumonia. For example, individuals with mutations in ] (BTK).<ref>Genetics in community-acquired Pneumonia : Current Opinion in Pulmonary Medicine. (2019). LWW. https://journals.lww.com/co-pulmonarymedicine/Abstract/2019/05000/Genetics_in_community_acquired_Pneumonia.17.aspx</ref> These mutations lead to the development of a disease called ] which inhibits the formation of white blood cells and mature ]. Without functional BTK, the development cycle of B lymphocytes is stopped at the pre-B cell stage which results in the loss of mature lymphocytes in the bone marrow and ]. In BTK, the mutation of arg525 to gln was particularly responsible for the lowered functionality of BTK.<ref>Chen, Shaw, Petty, North. (2020, December 11). Host genetic effects in Pneumonia. Cell. https://www.cell.com/ajhg/fulltext/S0002-9297(20)30446-8</ref> Due to decreased white blood cell count, this immune deficiency increases one’s susceptibility to Pneumonia greatly.
	] (]) 18:21, 12 September 2018 (UTC)
	::Would need a recommendation by say WHO. Also do we have a good ref to support bubble CPAP in pneumonia?
	::Seems like the trials are ongoing https://www.ncbi.nlm.nih.gov/pubmed/28883928 ] (] · ] · ]) 19:28, 12 September 2018 (UTC)
	:::This review says "More research is needed on the implementation, cost and effectiveness of CPAP in the management of pneumonia and in neonatal care in developing countries" https://www.ncbi.nlm.nih.gov/pubmed/24165032 ] (] · ] · ]) 19:33, 12 September 2018 (UTC)

			A broad ] study aimed at identifying ] associated with pulmonary function and Pneumonia in humans mapped 137 loci of interest. 116 of the 137 loci were found to be responsible for both pulmonary function and susceptibility to Pneumonia. <ref>Khadzhieva, Kuzovlev, Salnikova. (2019, December). Pneumonia: host susceptibility and shared genetics with pulmonary function and other traits. PubMed. https://pubmed.ncbi.nlm.nih.gov/31487037/</ref> Furthermore, 336/340 ] were shared between Pneumonia and pulmonary function <ref>Khadzhieva, Kuzovlev, Salnikova. (2019, December). Pneumonia: host susceptibility and shared genetics with pulmonary function and other traits. PubMed. https://pubmed.ncbi.nlm.nih.gov/31487037/</ref>. Genes that are responsible for pulmonary function play an integral role in overall lung development and inflammation pathways that are key to an immune response. Individuals with an impaired pulmonary function due to certain predisposed developmental, inflammatory, or cardiovascular traits were found to be at a higher risk for Pneumonia. This high degree of overlap of genes and the correlation between pulmonary function traits and susceptibility to Pneumonia could mean that there is a major genetic factor.
	== Semi-protected edit request on 15 October 2018 ==

			There are nine disease genes currently known to increase susceptibility to ]. Three of the disease genes are responsible for the production of ] by alveoli in the lungs: Surfactant protein C (SFTPC), Surfactant protein A2 (SFTPA2), and Adenosine triphosphate-binding cassette subfamily A member 3 (ABCA3).<ref>ILD Colaborative. (2020). Genetics of Familial Idiopathic Interstitial Pneumonia. https://www.ildcollaborative.org/resources/genetics-of-familial-idiopathic-interstitial-Pneumonia</ref> Surfactant is a mixture of proteins and fats that helps keep the ] from collapsing when a person exhales and also protects lung cells from infection. Mutations at these loci have age-dependent effects, however, it is not yet possible to predict which family members will develop Pneumonia at what age. The other six disease genes are associated with ]: Telomerase reverse transcriptase , Telomerase RNA component , Dyskerin , Telomere repeat binding factor 1-interacting nuclear factor 2 , Regulator of telomere elongation helicase , Poly(A)-specific ribonuclease . Mutations in these genes impede the ability of the body to repair damage in the telomeres of ] and thus they become shorter, which can also increase the risk of developing Pneumonia, amongst many other diseases.<ref>ILD Colaborative. (2020). Genetics of Familial Idiopathic Interstitial Pneumonia. https://www.ildcollaborative.org/resources/genetics-of-familial-idiopathic-interstitial-Pneumonia</ref>
	{{edit semi-protected\|Pneumonia\|answered=yes}}
			Currently, doctors do offer genetic testing for these 9 disease genes in cases where two or more individuals in the same family have Pneumonia.<ref>Kropski, YOung, Cogan, Mitchell, Lancaster, Worrell, Markin, Liu. (2017, June 1). Genetic Evaluation and Testing of Patients and Families with Idiopathic Pulmonary Fibrosis. National Library of Medicine. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5470751/</ref>
	{{dashboard.wikiedu.org assignment \| course = Misplaced Pages:Wiki_Ed/University_of_Wisconsin-Eau_Claire/Epidemiology_ENPH_450_(Fall_2018) \| reviewers = ] \| start_date = 2018-09-06 \| end_date = 2018-12-13 }}
	We would like to add Cochrane Evidence

			During the COVID-19 pandemic, patients infected with COVID-19 were found to have twice the risk of developing pneumonia.<ref>“DNA Test Can Quickly Identify Pneumonia in Patients with Severe COVID-19, Aiding Faster Treatment.” ScienceDaily, ScienceDaily, 15 Jan. 2021, https://www.sciencedaily.com/releases/2021/01/210115091340.htm.</ref> Doctors have developed a DNA test that uses multiple polymerase chain reactions to detect DNA and antibiotic resistance of in a patient’s blood sample. This can be done in the span of four hours which is much quicker than the conventional method of growing bacterial cultures which is prone to false negatives due to patients receiving antibiotics before the sample is collected. The test expedites the treatment procedure with doctors being able to prescribe antibiotics at an earlier stage and making it more effective in the treatment of Pneumonia.

			=== References ===
	Review : oral care for Nursing home acquired pneumoria 2018
			Chen, Shaw, Petty, North. (2020, December 11). Host genetic effects in Pneumonia. Cell. https://www.cell.com/ajhg/fulltext/S0002-9297(20)30446-8

			Genetics in community-acquired Pneumonia : Current Opinion in Pulmonary Medicine. (2019). LWW. https://journals.lww.com/co-pulmonarymedicine/Abstract/2019/05000/Genetics_in_community_acquired_Pneumonia.17.aspx
	Conclusion : there is no evidence that one approach to oral care is better than the other one prevening nursing home acquired pneumonia

			ILD Colaborative. (2020). Genetics of Familial Idiopathic Interstitial Pneumonia. https://www.ildcollaborative.org/resources/genetics-of-familial-idiopathic-interstitial-Pneumonia
	There is no high quality evidence about the topic.

			Khadzhieva, Kuzovlev, Salnikova. (2019, December). Pneumonia: host susceptibility and shared genetics with pulmonary function and other traits. PubMed. https://pubmed.ncbi.nlm.nih.gov/31487037/
	Liu, Chang; Cao, Yubin; Lin, Jie; Ng, Linda; Needleman, Ian; Walsh, Tanya; Li, Chunjie (2018). "Oral care measures for preventing nursing home‐acquired pneumonia". Cochrane Database of Systematic Reviews (9). doi:10.1002/14651858.CD012416.pub2. ISSN 1465-1858.

			Kropski, YOung, Cogan, Mitchell, Lancaster, Worrell, Markin, Liu. (2017, June 1). Genetic Evaluation and Testing of Patients and Families with Idiopathic Pulmonary Fibrosis. National Library of Medicine. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5470751/

			Waterer, Wunderink. (2010). Genetic susceptibility to Pneumonia. PubMed. https://pubmed.ncbi.nlm.nih.gov/15802163/
	```` ] (]) 14:16, 15 October 2018 (UTC)

			Guin, Debleena, et al. “Human Genetic Factors Associated with Pneumonia Susceptibility, a Cue for Covid-19 Mortality.” MedRxiv, Cold Spring Harbor Laboratory Press, 1 Jan. 2021, https://www.medrxiv.org/content/10.1101/2021.06.03.21258106v1.full.
	{{reflist-talk}}

			“DNA Test Can Quickly Identify Pneumonia in Patients with Severe COVID-19, Aiding Faster Treatment.” ScienceDaily, ScienceDaily, 15 Jan. 2021, https://www.sciencedaily.com/releases/2021/01/210115091340.htm.
	:] '''Not done:''' According to the page's protection level you should be able to ]. If you seem to be unable to, please reopen the request with further details.<!-- Template:ESp --> {{Ping\|CarloFirst}} I checked your user rights and according to your rights you should be able to edit this page on your own. If this is a ] edit request please use {{tlx\|request edit}} for conflict-of-interest edits. ''']] ]''' 15:24, 16 October 2018 (UTC)
			{{Reflist}} ] (]) 00:26, 11 May 2022 (UTC)
			: ] '''Not done:''' it's not clear what changes you want to be made. Please mention the specific changes in a "change X to Y" format and provide a ] if appropriate.<!-- Template:ESp --> —](]) 05:46, 14 May 2022 (UTC)

			== ] ==
	== Treatment or not? The Old Man's Friend ==

			On Misplaced Pages there are 5 links to "]" which then redirects here, yet "hypostatic" does not appear anywhere within this article. The disambiguation for ] only gives one medically relevant definition: "Hypostasis ''(])'', corpse's discoloration". That might be helpful, but it's for a different organ. I can only take an educated guess at what hypostatic pneumonia means, and I wouldn't be sure the right place to describe it if I knew. ] (]) 20:12, 26 September 2022 (UTC)
	Some articles about pneumonia as the old man's friend, allowing the frail and slowly painfully dying to slip away comparatively quickly and easily make the case that if someone would rather not continue suffering (or has not specified, but those close think they would prefer this) that the pneumonina should not be treated (with antibiotics), but rather the uncomfortable symptoms. Could someone creat a section dealing with this debate? ] (]) 03:24, 12 December 2018 (UTC)

			== ''Cause'' section should probably be updated to take into account the prevalence of SARS-CoV-2 amongst pneumoniae causes ==
	==Coloring on this X rays is not as good as the prior==
	]

			This is important because such beliefs may lead the public to not consiser SARS-CoV-2 as a potential cause of pneumonia and lead to unjustified intakes of antibiotics. ] (]) 06:09, 29 September 2023 (UTC)
	] (] · ] · ]) 05:36, 12 February 2019 (UTC)

			== Is pneumonia the same as asthma? ==
	== Semi-protected edit request on 24 March 2019 ==

			No, they're different as ] is a ] and can cause ] , but ] is a disease of the ]; filling the ] with a fluid and can result a cause of death, for more information, watch the ] video or search the article ]. Also the article ]
	{{edit semi-protected\|Pneumonia\|answered=yes}}
	] (]) 07:21, 24 ~~March~~ ~~2019~~ (UTC)		] (]) 11:20, 8 April 2024 (UTC)

	:] '''Not done:''' it's not clear what changes you want to be made. Please mention the specific changes in a "change X to Y" format and provide a ] if appropriate.<!-- Template:ESp --> ] (]) 17:03, 24 March 2019 (UTC)
			== Add A Fact: "Child dies of pneumonia every 43 seconds" ==
			{{atop
			\| result = {{nd}}—I will go out on a limb to say this is the type of statistic that generally tugs the heartstrings more than it meaningfully educates. It would be an unencyclopedic addition. <span style="border-radius:2px;padding:3px;background:#1E816F">]<span style="color:#fff"> ‥ </span>]</span> 03:10, 7 November 2024 (UTC)
			}}

			I found a fact that might belong in this article. See the quote below
			<blockquote>
			A child dies of pneumonia every 43 seconds
			</blockquote>
			The fact comes from the following source:
			: https://data.unicef.org/topic/child-health/pneumonia/


			Additional comments from user: It is alarming how these illnesses affect children, highlighting the urgent need to prevent and reverse the current situation.

			This post was generated using the ] browser extension.

			] (]) 16:37, 5 November 2024 (UTC)
			{{abot}}

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Semi-protected edit request on 10 May 2022 - Genetics of Pneumonia

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It has been found that vulnerability to Pneumonia and its severity may be linked to underlying genetic mechanisms. These mechanisms, such as the CYP1A1 gene (CYP1A1) regulating inflammatory responses and sepsis, can influence the efficacy of the immune system which can either be detrimental or beneficial in combating Pneumonia.

Individuals with certain genetic variants can be at risk for a higher susceptibility to Pneumonia. For example, individuals with mutations in Bruton’s tyrosine kinase (BTK). These mutations lead to the development of a disease called X-linked agammaglobulinemia which inhibits the formation of white blood cells and mature B cells. Without functional BTK, the development cycle of B lymphocytes is stopped at the pre-B cell stage which results in the loss of mature lymphocytes in the bone marrow and lymphatic system. In BTK, the mutation of arg525 to gln was particularly responsible for the lowered functionality of BTK. Due to decreased white blood cell count, this immune deficiency increases one’s susceptibility to Pneumonia greatly.

A broad mapping study aimed at identifying loci associated with pulmonary function and Pneumonia in humans mapped 137 loci of interest. 116 of the 137 loci were found to be responsible for both pulmonary function and susceptibility to Pneumonia. Furthermore, 336/340 SNPs were shared between Pneumonia and pulmonary function . Genes that are responsible for pulmonary function play an integral role in overall lung development and inflammation pathways that are key to an immune response. Individuals with an impaired pulmonary function due to certain predisposed developmental, inflammatory, or cardiovascular traits were found to be at a higher risk for Pneumonia. This high degree of overlap of genes and the correlation between pulmonary function traits and susceptibility to Pneumonia could mean that there is a major genetic factor.

There are nine disease genes currently known to increase susceptibility to idiopathic interstitial Pneumonia. Three of the disease genes are responsible for the production of surfactant by alveoli in the lungs: Surfactant protein C (SFTPC), Surfactant protein A2 (SFTPA2), and Adenosine triphosphate-binding cassette subfamily A member 3 (ABCA3). Surfactant is a mixture of proteins and fats that helps keep the alveoli from collapsing when a person exhales and also protects lung cells from infection. Mutations at these loci have age-dependent effects, however, it is not yet possible to predict which family members will develop Pneumonia at what age. The other six disease genes are associated with telomeres: Telomerase reverse transcriptase , Telomerase RNA component , Dyskerin , Telomere repeat binding factor 1-interacting nuclear factor 2 , Regulator of telomere elongation helicase , Poly(A)-specific ribonuclease . Mutations in these genes impede the ability of the body to repair damage in the telomeres of chromosomes and thus they become shorter, which can also increase the risk of developing Pneumonia, amongst many other diseases. Currently, doctors do offer genetic testing for these 9 disease genes in cases where two or more individuals in the same family have Pneumonia.

During the COVID-19 pandemic, patients infected with COVID-19 were found to have twice the risk of developing pneumonia. Doctors have developed a DNA test that uses multiple polymerase chain reactions to detect DNA and antibiotic resistance of streptococcus pneumoniae in a patient’s blood sample. This can be done in the span of four hours which is much quicker than the conventional method of growing bacterial cultures which is prone to false negatives due to patients receiving antibiotics before the sample is collected. The test expedites the treatment procedure with doctors being able to prescribe antibiotics at an earlier stage and making it more effective in the treatment of Pneumonia.

References

Chen, Shaw, Petty, North. (2020, December 11). Host genetic effects in Pneumonia. Cell. https://www.cell.com/ajhg/fulltext/S0002-9297(20)30446-8

Genetics in community-acquired Pneumonia : Current Opinion in Pulmonary Medicine. (2019). LWW. https://journals.lww.com/co-pulmonarymedicine/Abstract/2019/05000/Genetics_in_community_acquired_Pneumonia.17.aspx

ILD Colaborative. (2020). Genetics of Familial Idiopathic Interstitial Pneumonia. https://www.ildcollaborative.org/resources/genetics-of-familial-idiopathic-interstitial-Pneumonia

Khadzhieva, Kuzovlev, Salnikova. (2019, December). Pneumonia: host susceptibility and shared genetics with pulmonary function and other traits. PubMed. https://pubmed.ncbi.nlm.nih.gov/31487037/

Kropski, YOung, Cogan, Mitchell, Lancaster, Worrell, Markin, Liu. (2017, June 1). Genetic Evaluation and Testing of Patients and Families with Idiopathic Pulmonary Fibrosis. National Library of Medicine. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5470751/

Waterer, Wunderink. (2010). Genetic susceptibility to Pneumonia. PubMed. https://pubmed.ncbi.nlm.nih.gov/15802163/

Guin, Debleena, et al. “Human Genetic Factors Associated with Pneumonia Susceptibility, a Cue for Covid-19 Mortality.” MedRxiv, Cold Spring Harbor Laboratory Press, 1 Jan. 2021, https://www.medrxiv.org/content/10.1101/2021.06.03.21258106v1.full.

“DNA Test Can Quickly Identify Pneumonia in Patients with Severe COVID-19, Aiding Faster Treatment.” ScienceDaily, ScienceDaily, 15 Jan. 2021, https://www.sciencedaily.com/releases/2021/01/210115091340.htm.

Guin, Debleena, et al. “Human Genetic Factors Associated with Pneumonia Susceptibility, a Cue for Covid-19 Mortality.” MedRxiv, Cold Spring Harbor Laboratory Press, 1 Jan. 2021, https://www.medrxiv.org/content/10.1101/2021.06.03.21258106v1.full.
Genetics in community-acquired Pneumonia : Current Opinion in Pulmonary Medicine. (2019). LWW. https://journals.lww.com/co-pulmonarymedicine/Abstract/2019/05000/Genetics_in_community_acquired_Pneumonia.17.aspx
Chen, Shaw, Petty, North. (2020, December 11). Host genetic effects in Pneumonia. Cell. https://www.cell.com/ajhg/fulltext/S0002-9297(20)30446-8
Khadzhieva, Kuzovlev, Salnikova. (2019, December). Pneumonia: host susceptibility and shared genetics with pulmonary function and other traits. PubMed. https://pubmed.ncbi.nlm.nih.gov/31487037/
Khadzhieva, Kuzovlev, Salnikova. (2019, December). Pneumonia: host susceptibility and shared genetics with pulmonary function and other traits. PubMed. https://pubmed.ncbi.nlm.nih.gov/31487037/
ILD Colaborative. (2020). Genetics of Familial Idiopathic Interstitial Pneumonia. https://www.ildcollaborative.org/resources/genetics-of-familial-idiopathic-interstitial-Pneumonia
ILD Colaborative. (2020). Genetics of Familial Idiopathic Interstitial Pneumonia. https://www.ildcollaborative.org/resources/genetics-of-familial-idiopathic-interstitial-Pneumonia
Kropski, YOung, Cogan, Mitchell, Lancaster, Worrell, Markin, Liu. (2017, June 1). Genetic Evaluation and Testing of Patients and Families with Idiopathic Pulmonary Fibrosis. National Library of Medicine. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5470751/
“DNA Test Can Quickly Identify Pneumonia in Patients with Severe COVID-19, Aiding Faster Treatment.” ScienceDaily, ScienceDaily, 15 Jan. 2021, https://www.sciencedaily.com/releases/2021/01/210115091340.htm.

Editor3456123123 (talk) 00:26, 11 May 2022 (UTC)

Not done: it's not clear what changes you want to be made. Please mention the specific changes in a "change X to Y" format and provide a reliable source if appropriate. —Sirdog (talk) 05:46, 14 May 2022 (UTC)

Hypostatic pneumonia

On Misplaced Pages there are 5 links to "hypostatic pneumonia" which then redirects here, yet "hypostatic" does not appear anywhere within this article. The disambiguation for hypostasis only gives one medically relevant definition: "Hypostasis (livor mortis), corpse's discoloration". That might be helpful, but it's for a different organ. I can only take an educated guess at what hypostatic pneumonia means, and I wouldn't be sure the right place to describe it if I knew. DAVilla (talk) 20:12, 26 September 2022 (UTC)

Cause section should probably be updated to take into account the prevalence of SARS-CoV-2 amongst pneumoniae causes

This is important because such beliefs may lead the public to not consiser SARS-CoV-2 as a potential cause of pneumonia and lead to unjustified intakes of antibiotics. MathieuSchopfer (talk) 06:09, 29 September 2023 (UTC)

Is pneumonia the same as asthma?

No, they're different as asthma is a difficulty for breathing and can cause tiredness , but pneumonia is a disease of the lungs; filling the alveolus with a fluid and can result a cause of death, for more information, watch the Videowiki/Pneumonia video or search the article Pneumonia. Also the article Asthma 112.198.178.96 (talk) 11:20, 8 April 2024 (UTC)

Add A Fact: "Child dies of pneumonia every 43 seconds"

Not done—I will go out on a limb to say this is the type of statistic that generally tugs the heartstrings more than it meaningfully educates. It would be an unencyclopedic addition. Remsense ‥ 论 03:10, 7 November 2024 (UTC)

The following discussion is closed. Please do not modify it. Subsequent comments should be made on the appropriate discussion page. No further edits should be made to this discussion.

I found a fact that might belong in this article. See the quote below

A child dies of pneumonia every 43 seconds

The fact comes from the following source:

https://data.unicef.org/topic/child-health/pneumonia/

Additional comments from user: It is alarming how these illnesses affect children, highlighting the urgent need to prevent and reverse the current situation.

This post was generated using the Add A Fact browser extension.

Laiasolagonzalez (talk) 16:37, 5 November 2024 (UTC)

The discussion above is closed. Please do not modify it. Subsequent comments should be made on the appropriate discussion page. No further edits should be made to this discussion. Categories: