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| The ] forms a capsule around the ] laterally. It is separated from the thalamus by the ]. |
The ] is part of the ventral thalamus that forms a capsule around the ] laterally. It is separated from the thalamus by the ]. Reticular cells are GABAergic, and have discoid dendritic arbors in the plane of the nucleus. Like the dorsal thalamic nuclei, the thalamic reticular nucleus receives input from the ] and and dorsal thalamic nuclei. Primary thalamic reticular nucleus efferent fibers project to other thalamic nuclei, but never to the cerebral cortex. This is the only thalamic nucleus that does not project to the cerebral cortex. | ||
| The function of the thalamic reticular nucleus is not understood, although it has some role in absence seizures(von Krosigk et al., 1993). Their is debate over the presence of distinct sectors within the nucleus that each correspond to a different sensory or cognitive modality. | |||
| For orginal connectivity anatomy see Jones 1975 | |||
| For disscusion of mapping and cross modality pathways see Crabtree 2002 | |||
Revision as of 04:04, 6 June 2005
The thalamic reticular nucleus is part of the ventral thalamus that forms a capsule around the thalamus laterally. It is separated from the thalamus by the external medullary lamina. Reticular cells are GABAergic, and have discoid dendritic arbors in the plane of the nucleus. Like the dorsal thalamic nuclei, the thalamic reticular nucleus receives input from the cerebral cortex and and dorsal thalamic nuclei. Primary thalamic reticular nucleus efferent fibers project to other thalamic nuclei, but never to the cerebral cortex. This is the only thalamic nucleus that does not project to the cerebral cortex.
The function of the thalamic reticular nucleus is not understood, although it has some role in absence seizures(von Krosigk et al., 1993). Their is debate over the presence of distinct sectors within the nucleus that each correspond to a different sensory or cognitive modality.
For orginal connectivity anatomy see Jones 1975 For disscusion of mapping and cross modality pathways see Crabtree 2002