Tricuspid valve stenosis - Misplaced Pages

(Redirected from Tricuspid stenosis) Type of valvular heart disease

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Medical condition

Tricuspid valve stenosis
Specialty	Cardiology

Tricuspid valve stenosis is a valvular heart disease that narrows the opening of the heart's tricuspid valve. It is a relatively rare condition that causes stenosis (increased restriction of blood flow through the valve).

Cause

Causes of tricuspid valve stenosis are:

Diagnosis

A mild diastolic murmur can be heard during auscultation caused by the blood flow through the stenotic valve. It is best heard over the left sternal border with rumbling character and tricuspid opening snap with wide-splitting S2. The diagnosis will typically be confirmed by an echocardiograph, which will also allow the physician to assess its severity.

Treatment

Tricuspid valve stenosis itself usually does not require treatment. If stenosis is mild, monitoring the condition closely suffices. However, severe stenosis, or damage to other valves in the heart, may require surgical repair or replacement.

The treatment is usually by surgery (tricuspid valve replacement) or percutaneous balloon valvuloplasty. The resultant tricuspid regurgitation from percutaneous treatment is better tolerated than the insufficiency occurring during mitral valvuloplasty.

References

"Problem: Tricuspid Valve Stenosis". www.heart.org. Retrieved 2017-10-31.
Ing, Frank; Sullivan, Patrick; Takao, Cheryl (2018). "Chapter 46 - Catheter Device Therapies for Heart Failure". Heart Failure in the Child and Young Adult. Academic Press. pp. 583–622. ISBN 978-0-12-802393-8. Retrieved 22 July 2020.

External links

Classification	D ICD-10: I07.0, I36.0, Q22.4 ICD-9-CM: 397.0, 746.9 MeSH: D014264
External resources	eMedicine: med/2315

Cardiovascular disease (heart)

Ischemia

Coronary disease	Coronary artery disease (CAD) Coronary artery aneurysm Spontaneous coronary artery dissection (SCAD) Coronary thrombosis Coronary vasospasm Myocardial bridge
Active ischemia	Angina pectoris Prinzmetal's angina Stable angina Acute coronary syndrome Myocardial infarction Unstable angina
Sequelae	hours Hibernating myocardium Myocardial stunning days Myocardial rupture weeks Ventricular aneurysm Dressler syndrome

Layers

Pericardium

Myocardium

Endocardium /
valves

Endocarditis	infective endocarditis Subacute bacterial endocarditis non-infective endocarditis Libman–Sacks endocarditis Nonbacterial thrombotic endocarditis
Valves	mitral prolapse stenosis regurgitation aortic stenosis regurgitation tricuspid stenosis regurgitation pulmonary stenosis regurgitation

Conduction /
arrhythmia

Bradycardia

Tachycardia
(paroxysmal and sinus)

Supraventricular	Atrial Multifocal Junctional AV nodal reentrant Junctional ectopic
Ventricular	Accelerated idioventricular rhythm Catecholaminergic polymorphic Torsades de pointes

Premature contraction

Pre-excitation syndrome

Flutter / fibrillation

Other / ungrouped

Other

Congenital heart defects

Heart septal defect

Aortopulmonary septal defect	Double outlet right ventricle Taussig–Bing syndrome Transposition of the great vessels dextro levo Persistent truncus arteriosus Aortopulmonary window
Atrial septal defect	Sinus venosus atrial septal defect Lutembacher's syndrome
Ventricular septal defect	Tetralogy of Fallot
Atrioventricular septal defect	Ostium primum
Consequences	Cardiac shunt Cyanotic heart disease Eisenmenger syndrome

Valvular heart disease

Right	pulmonary valves stenosis insufficiency absence tricuspid valves atresia regurgitation stenosis Ebstein's anomaly
Left	aortic valves stenosis insufficiency bicuspid mitral valves stenosis regurgitation